By Mary Sheppard
The second one variation of Practical Cardiovascular Pathology offers a vital textual content and color atlas assisting all basic pathologists to spot quite a lot of cardiovascular stipulations speedily and thoroughly in either their diagnostic and post-mortem work.
With many new stipulations defined, and diverse new illustrations, the second one version of this highly-acclaimed textual content will remain priceless to all pathologists and trainees.
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Additional resources for Practical Cardiovascular Pathology, 2nd edition
The mortality rate in the acute stage is high and even surgical repair is high risk, with large infarcts showing little viable tissue to work on for repair. 56 Late ventricular rupture in myocardial infarction. Viewed from the epicardial surface there is a localized external bulge with a rupture point at its apex. Papillary muscle rupture The majority of the complications listed so far are directly related to transmural large infarcts. Papillary muscle rupture is an exception in that the infarcts can be small and may not be transmural.
While formerly regarded as a passive precipitation of calcium phosphate crystals, plaque calcification is now recognized as a regulated process in which osteopontin, osteonectin and osteocalcin are involved. Both macrophages and smooth muscle cells are involved in the production of these bone-promoting substances. Evidence of osteoclast- and osteoblast-like differentiation occurs in plaques and bone may be laid down. The question is whether calcification plays any more major role than hindering dissection by surgeons and pathologists.
Under these circumstances previously existing small vessels enlarge and allow collateral flow. 25 Eccentric lipid-rich coronary plaque. The plaque has a very large lipid core which is extending up close to the lumen with a very thin cap in one area. The lipid was soft and could be easily expressed from the plaque by presssure. Opposite the plaque is a segment of normal arterial wall. 26 Eccentric lipid rich plaque. This plaque has a large crumbling lipid core but there is more collagenous fibrous tissue and the cap is thick.
Practical Cardiovascular Pathology, 2nd edition by Mary Sheppard